1.Hilarious Paper #1 "Survival of acute hypernatremia due to massive soy sauce ingestion"...A 19-year-old man presented to the Emergency Department in a comatose state with seizure-like activity 2 hours after ingesting a quart of soy sauce. He was administered 6 L of free water over 30 min and survived neurologically intact without clinical sequelae. Corrected for hyperglycemia, the patient's peak serum sodium was 196 mmol/L, which, to our knowledge, is the highest documented level in an adult patient to survive an acute sodium ingestion without neurologic deficits..."
2. Hilarious Paper #2:"Misidentification of vagus nerve stimulator for intravenous access and other major adverse events." We reviewed our experience with major adverse events, after accidental puncture of a stimulator wire by an emergency room physician seeking intravenous access to treat status epilepticus..."
3. Ebstein's anomaly is the most fascinating congenital heart defect: 
-the tricuspid valve is displaced downwards (valves in a sense "fused" to ventricular wall, fail to separate adequately to "rise" up to normal position)
-tricuspid ends up being severely regurgitant, from a combination of being displaced and atrial enlargement.
-why Ebstein's is associated with cyanosis: increased pulmonary resistance at birth (and for first few months of life) means that blood from the RV preferentially regurgitates back to RA rather than going into pulmonary vasculature. Blood from RA shunts R to L through PFO, into LA, LV, through aorta, and then back through PDA (since resistance in pulm vasculature is lower than systemic), and then because resistance in the RV is lower than pulm, it regurgitates back into RV, and then back into RA, in this unique circular shunt.
-this circular shunt is only in place while the PA resistance is high, i.e. in the first months of life. Thus, it has a high perinatal mortality, but once a child ages out of this phase, s/he can live a healthy and asymptomatic life.
-Ebstein's is frequently associated with wolff-parkinson-white
4. Transposition and TAPVR are not ductal-dependent congenital heart defects, they're PFO dependent. In these two defects, there are 2 separate circulations that are not communicating, and the way to fix it is via mixing of blood in the chambers, which a PDA will not do. PDA will only shunt blood from the higher to lower pressure sides, which is useless in this case. A PFO will ensure good admixing, since the atria are both low pressure systems. If the PFO is closing, an emergent balloon atrial septostomy is necessary.
5. Duct-dependent lesions are those where there is severe outflow obstruction from either the R or the L, due to severe stenosis (TOF, bad pulm or aortic stenosis, bad preduct coarctation) or lack of propulsion-- i.e. hypoplastic L or R heart (tricuspid atresia). In other words, lesions where it would be useful to shunt large amounts of blood mostly unidirectionally from systemic to pulmonary systems (or vice-versa).
6. Cyanotic heart lesions are those that have a R => L shunt, which requires (1) pulmonary outflow obstruction and (2) a shunt so blood goes to L side instead. Non-cyanotic heart lesions are those that have a L => R shunt, or those that have outflow obstruction but no shunt/admixing (i.e. pulm stenosis without VSD/ASD/PFO)
7. Refeeding syndrome: when someone has been in starvation for a long time, body stops making insulin; when it gets fed again, insulin goes up, which can cause electrolyte abnormalities:
-decreased phosphate, since the increased generation of ATP will lead to cellular uptake of blood phosphate. People can drop their phosphates very low, leading to bradycardia, lethargy.
-decreased calcium, which can lead to convusions, arrhythmias, tetany, numbness.
-decreased potassium, which can lead to arrhythmias. Insulin leads to K uptake
-decreased mag
8. The murmur of a VSD is not heard until a few months of life, because the increased pulmonary resistance means that there is little shunting of blood (L->R) across it, even though there is a pressure differential.
9. Acrocyanosis is normal in newborns and infants, can be thought of as a "physiologic reynaud's" as the body is still learning how to autonomically regulate heat and perfusion.
10. If you are attacked by a polar bear and are out of options, try shining your iphone light into its eyes to scare it off. [NPR] Disclaimer: this is definitely the worst idea ever, and will not work